Search results for "Retinyl Esters"

showing 10 items of 10 documents

Familial hypobetalipoproteinemia due to apolipoprotein B R463W mutation causes intestinal fat accumulation and low postprandial lipemia

2008

Abstract Objective Familial hypobetalipoproteinemia (FHBL) is characterized by inherited low plasma levels of apolipoprotein B (apoB)-containing lipoproteins. In this paper we investigated whether the already described APOB R463W missense mutation, a FHBL mutation able to impair the activity of microsomal triglyceride transfer protein (MTP), may cause intestinal fat accumulation and reduced postprandial lipemia. Methods Four out of five probands harboring APOB R463W mutation were compared with six healthy controls and six patients with celiac disease (CD). An oral fat load supplemented with retinyl palmitate (RP) was administered and a gastro-duodenal endoscopy with biopsy was performed. Re…

AdultMaleRetinyl Estersmedicine.medical_specialtySettore MED/09 - Medicina InternaAdolescentApolipoprotein BMutation MissenseapolipoproteinBlood lipidsHyperlipidemiasIntra-Abdominal FatBiologyMicrosomal triglyceride transfer proteinchemistry.chemical_compoundRetinyl palmitateInternal medicinemedicineHumansMissense mutationIntestinal MucosaChildVitamin ATriglyceridesApolipoproteins BTriglycerideMiddle AgedLipid MetabolismPostprandial Periodmedicine.diseasePostprandialEndocrinologychemistryHypobetalipoproteinemia Familial Apolipoprotein BB R463Wbiology.proteinFemalelipids (amino acids peptides and proteins)HypobetalipoproteinemiaDiterpenesCarrier ProteinsCardiology and Cardiovascular MedicineAtherosclerosis
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Low-density lipoproteins generated during an oral fat load in mild hypertriglyceridemic and healthy subjects are smaller, denser, and have an increas…

2005

Triglyceride-rich lipoproteins generated during the postprandial phase are atherogenic. Large very low-density lipoproteins (LDLs) or chylomicrons (CMs) are not as atherogenic as their remnants (Rem). Small and dense LDLs are associated with cardiovascular disease. Low-density lipoprotein size is partly under genetic control and is considered as a relatively stable LDL feature. In this article, we present data on retinyl palmitate kinetics correlated with the modification of LDL features in terms of size, density, and in vitro receptor binding affinity after an oral fat load. Six nondiabetic, hypertriglyceridemic (HTG) patients and 6 healthy controls were examined. Low-density lipoprotein s…

AdultMalemedicine.medical_specialtyRetinyl EstersSettore MED/09 - Medicina InternaEndocrinology Diabetes and MetabolismBinding CompetitiveModels Biologicalchemistry.chemical_compoundEndocrinologyInternal medicineRetinyl palmitateCell Line TumorChylomicronsmedicineHumansReceptorVitamin AHypertriglyceridemiaLow-density lipoproteins hypertriglyceridemia Fasting and postprandial LDLsTriglycerideCatabolismChemistryFastingFibroblastsPostprandial PeriodDietary FatsLipidsLipoproteins LDLKineticsEndocrinologyPostprandialReceptors LDLlipids (amino acids peptides and proteins)Density gradient ultracentrifugationElectrophoresis Polyacrylamide GelFemaleDiterpenesUltracentrifugationLipoproteinChylomicronMetabolism: clinical and experimental
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In vivo studies of altered expression patterns of p53 and proliferative control genes in chronic vitamin A deficiency and hypervitaminosis

2003

Several clinical trials have revealed that individuals who were given beta-carotene and vitamin A did not have a reduced risk of cancer compared to those given placebo; rather, vitamin A could actually have caused an adverse effect in the lungs of smokers [Omenn, G.S., Goodman, G.E., Thornquist, M.D., Balmes, J., Cullen, M.R., Glass, A., Keogh, J.P., Meyskens, F.L., Valanis, B., Williams, J.H., Barnhart, S. & Hammar, S. N. Engl. J. Med (1996) 334, 1150-1155; Hennekens, C.H., Buring, J.E., Manson, J.E., Stampfer, M., Rosner, B., Cook, N.R., Belanger, C., LaMotte, F., Gaziano, J.M., Ridker, P.M., Willet, W. & Peto, R. (1996) N. Engl. J. Med. 334, 1145-1149]. Using differential display techniq…

Cyclin-Dependent Kinase Inhibitor p21VitaminRetinyl Estersmedicine.medical_specialtyMacromolecular SubstancesProto-Oncogene Proteins c-junReceptors Retinoic AcidBlotting WesternRetinoic acidBiologymedicine.disease_causeBiochemistrychemistry.chemical_compoundIn vivoCyclinsInternal medicinemedicineAnimalsHypervitaminosis ARNA MessengerRats WistarVitamin AReceptorLungDifferential displayReverse Transcriptase Polymerase Chain ReactionVitamin A DeficiencyGene Expression ProfilingDNAmedicine.diseaseHypervitaminosisPrecipitin TestsRatsVitamin A deficiencyEndocrinologyGene Expression RegulationLiverchemistryChronic DiseaseImmunologyDiterpenesTumor Suppressor Protein p53CarcinogenesisCell DivisionEuropean Journal of Biochemistry
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Sensitive Analysis of Retinyl Esters by Isocratic Adsorption Chromatography

1989

A sensitive method for the determination of retinyl esters, including their geometric isomers, by isocratic adsorption HPLC is described. The development of a special recycling system allows the separation of all-trans-, 13-cis, 11-cis- and 9-cis-retinyl palmitate, -stearate, -oleate, -palmitoleate and -linoleate with short retention times and high sensitivity. Extraction of the retinyl esters from various organs with mobile phase avoids additional evaporation steps. The method was evaluated by the determination of retinyl esters in small tissue samples such as tongue, trachea and inner ear. This is the first report of the detection of retinyl esters in these tissues, which are known to dep…

MaleVitaminChromatographyGuinea PigseducationBiochemistry (medical)Clinical BiochemistryExtraction (chemistry)General MedicineHigh-performance liquid chromatographyRetinyl esterschemistry.chemical_compoundSpectrometry FluorescenceAdsorptionLiverchemistryStearateRetinyl palmitateAnimalsSpectrophotometry UltravioletTissue DistributionVitamin ALungChromatography High Pressure LiquidCis–trans isomerismClinical Chemistry and Laboratory Medicine
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Retinol and retinyl esters in pigment epithelium of rats with inherited retinal degeneration

1976

A comparative study of the retinol and retinyl ester concentrations was performed in the retinal pigment epithelium of the normal and affected rats. Our findings indicate that in dystrophic rat retinol content increases, whereas the amount of retinyl esters is always lower than normal. An hypothesis can be made on the deficiency of enzymic activities which regulate retinol retinol and retinyl esters levels in the pigment epithelium.

Retinal degenerationgenetic structuresRetinyl estersCellular and Molecular NeurosciencePigmentchemistry.chemical_compoundmedicineAnimalsPigment Epithelium of EyeVitamin AMolecular BiologyPharmacologyRetinal pigment epitheliumChemistryRetinal DegenerationAge FactorsRetinolCell Biologymedicine.diseaseEpitheliumRatsmedicine.anatomical_structureBiochemistryvisual_artvisual_art.visual_art_mediumMolecular MedicineExperientia
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An ex vivo model of the rat trachea to study the effect of inhalable toxic compounds

1996

Different cell culture and organ systems are used to evaluate the physiological responses of the airways to the effects of carcinogenic [e.g., benzo(a)pyrene] and anticarcinogenic (e.g., retinoids) compounds on cellular growth and differentiation. However, in contrast to in vivo conditions dissociated epithelial cells or tracheal ring cultures are covered with medium. Therefore, we developed an ex vivo perfusion model enabling evaluation of morphology and metabolism of different compounds under near-physiological conditions. The trachea was surrounded with culture medium and perfused with air by means of a small animal respirator. To test the viability of the system under various experiment…

Retinyl EstersOligosaccharidesBiologyCell morphologyOrgan cultureXenobioticschemistry.chemical_compoundOrgan Culture TechniquesIn vivoLectinsAnimalsBenzopyrenesRats WistarVitamin ACarcinogenVitamin A DeficiencyGeneral MedicineRatsTracheaMicroscopy ElectronBenzo(a)pyrenechemistryBiochemistryCell culturePyreneDiterpenesEx vivoProtein BindingResearch in Experimental Medicine
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Contribution of vitamin A to the oxidation resistance of human low density lipoproteins.

1995

This study investigated the antioxidant contribution of vitamin A in protecting human low density lipoprotein (LDL) against copper-stimulated oxidation. The presence of small amounts of retinol (0.033 ± 0.012 nmol/mol LDL) and retinyl palmitate (0.036 ± 0.021 nmol/mol LDL) was routinely ascertained in the LDL. A single oral supplementation with 20,000 IU vitamin A caused a two- to three-fold increase of retinol and retinyl palmitate in the LDL isolated 8 h after the supplementation. In comparison to autologous-control LDL, vitamin A-enriched LDL were more resistant to oxidation, as expressed both by a clear delay in the onset of lipid peroxidation and by a reduction of the rate of conjugate…

VitaminAdultMaleRetinyl EstersAntioxidantFree RadicalsArteriosclerosismedicine.medical_treatmentRetinyl esterIn Vitro Techniquesmedicine.disease_causeBiochemistryAntioxidantsLDLLipid peroxidationchemistry.chemical_compoundFree radicalIn vivoPhysiology (medical)Retinyl palmitatemedicineHumansVitamin ARetinolRetinolMiddle AgedLipoproteins LDLKineticsOxidative StresschemistryBiochemistryAtherosclerosiLow-density lipoproteinlipids (amino acids peptides and proteins)FemaleLipid PeroxidationAntioxidantDiterpenesOxidation-ReductionOxidative stressCopperFree radical biologymedicine
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Vitamin A in parenteral nutrition: uptake and distribution of retinyl esters after intravenous application.

1989

Short-term parenteral application of vitamin A was examined in rats. Retinyl margarinate, which is chemically similar to physiological retinyl esters, was used in vitamin A-depleted rats to study uptake, distribution, and storage of retinyl esters in tissues. Vitamin A-depleted and Vitamin A-sufficient rats were infused with a micellar suspension of retinyl margarinate for 7 h and then killed at different times. Retinyl margarinate was directly taken up by all tissues examined. It appears that infusion of retinyl esters in micellar form provides a direct way to supply vitamin A to peripheral, vitamin A-dependent tissues. Therefore, a short-term infusion of retinyl esters with an emulsifier …

VitaminMalemedicine.medical_specialtyRetinyl EstersMedicine (miscellaneous)Retinyl esterschemistry.chemical_compoundPharmacokineticsInfusion ProcedureInternal medicineTestismedicineDistribution (pharmacology)AnimalsTissue DistributionIntestinal MucosaVitamin ALungChromatography High Pressure LiquidChromatographyNutrition and Dieteticsbusiness.industryVitamin A DeficiencyRetinolMetabolismRatsTracheaEndocrinologyParenteral nutritionchemistryLiverParenteral Nutrition TotalDiterpenesbusinessSpleenThe American journal of clinical nutrition
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Long-Term Administration of High Dose Vitamin A to Rats Does Not Cause Fetal Malformations: Macroscopic, Skeletal and Physicochemical Finds

1996

A rat model was used to investigate whether high oral doses of vitamin A lead to fetal malformations and to what extent retinyl esters (RES) are transferred from the mother to the fetuses. Retinol and RES concentrations in plasma behave similarly in rats and humans. When high concentrations of vitamin A are administered, plasma retinol concentrations remain relatively constant, whereas plasma RES increased in parallel with the dose. To achieve an elevation from approximately 150 to > 1525 nmol x L(-1) in the experimental group before mating, female Ibm: RORO (spf) rats were fed a maintenance diet enriched with 15.2 x 10(3) retinol equivalents (RE) x kg(-1) at the start and increased stepwis…

VitaminRetinyl Estersmedicine.medical_specialtyChemical PhenomenaRetinoic acidMedicine (miscellaneous)Biologychemistry.chemical_compoundPregnancyOral administrationInternal medicinemedicineAnimalsVitamin AMaternal-Fetal ExchangeChromatography High Pressure LiquidFetusNutrition and DieteticsChemistry PhysicalRetinolAbnormalities Drug-InducedRetinol EquivalentEstersTeratologyRatsEndocrinologychemistryToxicityFemaleDiterpenesThe Journal of Nutrition
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Retinoids induce MMP-9 expression through RARalpha during mammary gland remodeling.

2007

Retinoic acid (RA) is a signaling molecule in the morphogenesis of the mammary gland, modulating the expression of matrix metalloproteinases (MMPs). The aim of this paper was to study the role of RA during weaning, which consists of three events: apoptosis of the secretory cells, degradation of the extracellular matrix, and adipogenesis. CRABP II and CRBP-1 carrier proteins increased significantly during weaning compared with lactating glands but reverted to control values after the litter resuckled. The effects of RA are mediated by the nuclear receptors RARalpha, RARbeta, RARgamma, and RXRalpha, which underwent an increase in protein levels during weaning. In an attempt to elucidate the R…

medicine.medical_specialtyRetinyl EstersTime FactorsPhysiologymedicine.drug_classReceptors Retinoic AcidEndocrinology Diabetes and MetabolismMammary glandMorphogenesisRetinoic acidApoptosisTretinoinWeaningMatrix metalloproteinaseBiologyStromelysin 1chemistry.chemical_compoundRetinoidsMammary Glands AnimalPregnancyPhysiology (medical)Internal medicinemedicineAnimalsLactationInvolution (medicine)RetinoidRNA MessengerRats WistarVitamin AMammary gland involutionAdipogenesisRetinoic Acid Receptor alphaRetinol-Binding Proteins CellularMatrix MetalloproteinasesExtracellular MatrixRatsRetinol-Binding Proteinsmedicine.anatomical_structureEndocrinologychemistryMatrix Metalloproteinase 9Matrix Metalloproteinase 2FemaleDiterpenesSignal TransductionAmerican journal of physiology. Endocrinology and metabolism
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